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possible OCD treatment

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Re: possible OCD treatment

Postby visualizations » Mon Jun 11, 2012 7:19 pm

I have been reading about ibogaine since october, and have heard some things about it before that.

It seems to be a good candidate for OCD because of several factors. The theraputic session involved which is basically locating the possible underlying causes of the unfolding of events. It is a very potent SSRI that lasts for months possibly. The metabolites stay in the body afterwards leaving one happier if they have been going through an existential crisis. It resensitizes a complex combination of receptors, so the brain could function at a better rate instead of having to take drugs or otherwise having chronic fatigue throughout the day. During the session dopamine is lowered since DOPAC (its metabolite) is increased by 50%. Two weeks after the session DOPAC decreases leaving one with more vitality. It increases brain neuroplasticity. There is a chance that the experience won't be loopy because of some of these factors.

Of course things could also go wrong, for example some people having to face harsh things then regretting the situation. Also the pharmacology interfering with other types of mental illness. It does seem to antagonize NMDA, so someone with cognitive dysfunction should be really informed on how to get that up to speed.

I haven't done it, but I have tried ayahuasca for theraputic purposes. It does seem to have a "gaian mother morality" instilled where several things are cleansed for the body, and it does seem to decrease depression. It doesnt seem to really get to the bottom of a psychological complex because ayahuasca is about going "outwards" in to the different symbolisms of historical dimensions of earth. While on the contrary, ibogaine is about going "inwards", zooming in on the essence of a person's life path. Ibogaine seems to be more of a "father" type morality which is located within the person. Kind of showing them the truth about the matter, it seems to have ecstatic points and negative points. The ibogaine experience from asking others doesnt seem to be radically more freustrating than an ayahuasca experience. There seems to be a quiet observer type effect, so the entire mind is not immersed in deep agony. So far I think the harshness of the experience may be scary for some because they are not experienced with those types of states, but it seems to be overblown. At first there is discomfort, nausea, regretableness but this is eventually filled with substance. Then later on it is filled with awe.

It does seem to be possible to bypass the dream-induced experience if one is leary of those types of experiences. Usually by taking repeated low doses over long periods of time, or low-middle-range doses with something like a low dose benzodiazepine, piracetam or other psychedelia dampening agents. This would help reset the neurochemistry in the background while simply giving a low loss of coordination, and stimulated feeling. At low enough doses it has no psychoactive effect. Yet, a flood dose is recommended to get the full digestion in one go. It is highly recommended to not take anything else with ibogaine, but low doses with low doses shouldnt hurt if they are not contraindicated.

Someone posted their experience with aniracetam/piracetam for a mild hair obsession, and there are also some other OCD ideas here:
http://www.longecity.org/forum/topic/16 ... s-for-ocd/


Yeah. The reason my brain went downhill after getting off the medicine could be some kind of psychological thing because I kept the love for the person inside me for 7 months. Mostly it was because getting off the medicine activated a "brain trauma" type mechanism which eventually always leads to depression. So that is why I have been trying very hard to get my brain up to speed, and these last two months have been accelerated. Well its pretty difficult to antagonize 7 different important receptors and come out top notch it feels like its going to make it for a month or two then it figures out that its depleted. So that is more of a medicinal brain trauma regimen, but it could also enhance a more thorough slow build up in increasing overall brain function.

For the OCD thing being genetic I would agree that my mental illness probability is associated with DISC1. Since 2004 I have been prone to love obsession for many years. The first time I couldnt really talk to the person. The second time I tried talking to the person, but they ended up being incompatible in a similar fashion. In numerological terms this is the 7 - 11 bridge. Which are opposite archetypes, basically they search for the lower more concrete truths while the 11s search for more spiritual truths. Or in some simple astrological terms this is earth/air (me) versus fire/water in contradicting fashions. Well they were both fire signs, and i'm a taurus so its hard. Since I have no fire at all. I'm trying to change the entire way that i'm wired to react and participate in "loving" others because so far it has caused me much pain instead of gain.

What I think about mental illness pharmacology is that there are a plethora of stabalizing and destabalizing genes available. The genes cannot directly be manipulated yet. What needs to happen at its core is enviornmental-genetic stimulation through medicines so that the genetic expression for "abnormal" genes is lessened, and the genetic expression for more modulatory genes are accented. This can be done by trying to understand the several layers of the brain from something as universal as calcium channel neurotransmission and cytoskeletal formation. Or the archiving of phospolipids in cell compounds. If the general matrix of receptor formation or neurotransmission is swayed in to neurodiversity by these special genes, there is always something that can at least help to sway the brain system back. A core process in neuroplasticity if the conditions are set towards the right direction is always BDNF (brain neurotropic factor) and at a more global level IGF-1. At least if the person is young with stem cell availablity and good enough metabolism, I'm a proponent of moderate levels of synaptic plasticity being available in several regions of the brain. For example, there was a case where the conditions were right, the person had brain damage so the brain itself migitated an entire subsystem to the opposite hemisphere of the brain where it could function. This is of course something that doesn't happen often, and there are still some neuroscientists that only believe in SVT and hippocampus neurogenesis. With no like prefrontal cortex neurogenesis. Even if there is no neurogensis in the area, receptor binding densities within the platform could be potentiated as well as dendritic extensions to multiple correlating regions could still be stimulated over time. The entire research in to epi-genetics and brain trauma is something that should be looked in to more often by doctors for the insight of possibilities in treating lesser mental diseases.
visualizations
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